“Turns out that in spite of the late-night pizza binges and countless bags of Doritos they consume, the average marijuana user is actually less likely to be obese than the general, non-pot-smoking public.”
“Processed Meat Raises Risk Of Dying From Cancer And Heart Disease, Study Finds” http://www.huffingtonpost.com/2013/03/07/processed-meat-cancer-heart-disease-death-risk_n_2829092.html
“Chicago ‘Hot Dogs Cause Butt Cancer… Meat Under Fire for Causing Cancer, Death” http://abcnews.go.com/Health/chicago-hot-dogs-butt-cancer-billboard-highlights-colon/story?id=15920567
“Bad diet a significant cause of preventable cancer” http://www.ingredientsnetwork.com/news-content/full/bad-diet-a-significant-cause-of-preventable-cancer
“Processed meat ‘linked to pancreatic cancer'” http://www.bbc.co.uk/news/health-16526695
“Red meat increases death, cancer and heart risk, says study” http://www.bbc.co.uk/news/health-17345967
“Cancer risk from BBQ meat. Tips for safer and healthier grilling, from the Harvard Health Letter” http://www.health.harvard.edu/press_releases/cancer-risk-from-bbq-meat
“Scientists warn consumption of processed meat linked to cancer” http://www.guardian.co.uk/science/2009/jun/15/processed-meat-bacon-bowel-cancer
“Drinking Lots Of Milk Can Cause Ovarian Cancer” http://news.peacefmonline.com/health/201307/169595.php
“The endocannabinoid system is an ancient lipid signaling network which in mammals modulates neuronal functions, inflammatory processes, and is involved in the aetiology of certain human lifestyle diseases, such as Crohn’s disease, atherosclerosis and osteoarthritis.
The system is able to downregulate stress-related signals that lead to chronic inflammation and certain types of pain, but it is also involved in causing inflammation-associated symptoms, depending on the physiological context.
The cannabinoid type-2 (CB2) receptor, which unlike the CB1 receptor does not induce central side effects, has been shown to be a promising therapeutic target. While CB1 receptor antagonists/inverse agonists are of therapeutic value, also CB2 receptor ligands including agonists are of pharmacological interest.
Although the endocannabinoid system is known to be involved in the regulation of energy homoeostasis and metabolism (mainly via CB1 receptors) there was hitherto no direct link between food intake and cannabinoid receptor activation. Our recent finding that beta-caryophyllene, a ubiquitous lipohilic plant natural product, selectively binds to the CB2 receptor and acts as a full agonist is unexpected…
In the case of the dietary natural product beta-caryophyllene, a full CB2 receptor-selective agonist in vitro, potent anti-inflammatory cannabimimetic effects are observed. Intriguingly, the lowest oral dose tested (5 mg/Kg) of this widespread and apparently non-toxic compound, which is also an FDA-approve food additive, was the most effective. Maybe this strengthens the hypothesis that beta-caryophyllene is indeed a dietary cannabinoid, thus inferring that by eating this compound the endocannabinoid system may be modulated in a beneficial way…”
“Endocannabinoid signalling participates in the control of neurogenesis, especially after brain insults. Obesity may explain alterations in physiology affecting neurogenesis, although it is unclear whether cannabinoid signalling may modulate neural proliferation in obese animals. Here we analyse the impact of obesity by using two approaches, a high-fat diet (HFD, 60% fat) and a standard/low-fat diet (STD, 10% fat), and the response to a subchronic treatment with the cannabinoid receptor type 1 (CB1) inverse agonist AM251 (3 mg/kg) on cell proliferation of two relevant neurogenic regions, namely the subventricular zone in the striatal wall of the lateral ventricle (SVZ) and the subgranular zone of the dentate gyrus (SGZ), and also in the hypothalamus given its role in energy metabolism. We found evidence of an interaction between diet-induced obesity and CB1 signalling in the regulation of cell proliferation. AM251 reduced caloric intake and body weight in obese rats, as well as corrected plasma levels of cholesterol and triglycerides. AM251 is shown, for the first time, to modulate cell proliferation in HFD-obese rats only. We observed an increase in the number of 5-bromo-2-deoxyuridine-labelled (BrdU+) cells in the SGZ, but a decrease in the number of BrdU+ cells in the SVZ and the hypothalamus of AM251-treated HFD rats. These BrdU+ cells expressed the neuron-specific βIII-tubulin. These results suggest that obesity may impact cell proliferation in the brain selectively, and provide support for a role of CB1 signalling regulation of neurogenesis in response to obesity.”
“You know the stereotype: Marijuana user = Jonah Hill-looking dude who lives on his friend’s couch.
But if you look at the daily smokers around you, and we know you have daily smokers around you, this being the pot shop capital of the nation and all, what are you looking at?
You see skinny people.
For the first time, a study seems to back up the observation that stoners are actually lithe
Research published last week in the American Journal of Epidemiology (via our friends at SF Weekly) finds that weed smokers on average are more likely to have normal weight.
Parsing data, researchers found that 22 percent of nonsmokers were overweight while only 14 percent of bud aficionados were. Those ID’d as potheads had to smoke at least three times a week under the study.
What munchies? This might explain why pot-smoking women are often, well, smoking hot.
Interesting, because one of the arguments for legalizing medical marijuana is that it can be used to boost the appetites of AIDS sufferers and cancer patients who have trouble keeping weight on.
Still a valid use? Whatever works.
But we do know that the Marijuana Diet sounds way cooler than any eating plan South Beach Miami has ever produced.”
[@dennisjromero/djromero@laweekly.com]
http://blogs.laweekly.com/informer/2011/09/marijuana_skinny_fat_study.php